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If you have found what you are looking for on this site, and you are a copyright owner, and want any article or info on this site removed, or you want your images or articles on this site removed, you can simply e-mail us.Oxidative stress in the pathophysiology of the cardiovascular system.
The thiol antioxidant GSH has many actions beyond its role as a direct scavenger of reactive oxygen species (ROS). The metabolism of GSH is complex, including conversion to cysteine and sulfenic acid, glutathione disulfide formation, and the reaction of glutathione sulfhydryl groups to sulfenic acid, via cysteine adduct formation. However, under physiological conditions, GSH represents a substantial defense system against ROS. The rate of ROS production in the myocardium is approximately 2 to 5 mM ROS/min, and the rate of ROS consumption is approximately 25 to 45 mM GSH/min. However, oxidative stress can be produced in the myocardium by exogenous sources as well as endogenous sources, including inflammation, hypoxia/anoxia, and
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